Oxidative stress cisplatin resistance

keywords: cisplatin resistance; epithelial ovarian cancer; reactive oxygen species. publication types. research support, n.ı.h., extramural. mesh terms.

ros

09.03. cisplatin is commonly utilized in the treatment of solid tumors. ıts mechanism of action is complex and multiple mechanisms of resistance .

cisplatin resistance reversal in lung cancer by tumor acidity

09.02.2022 cisplatin resistance hinders the improvement of the prognosis of patients with in oxidative stress and cisplatin response of ovarian cancer.

failure of elevating calcium ınduces oxidative

herein, we develop a novel strategy to overcome cisplatin drug resistance through human lung cancer cells a549r under amplified oxidative stress usi.

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cisplatin is the firstline chemotherapy drug for the clinical treatment of growth and cisplatin resistance ın vitro via activating oxidative stress .

reduction in mitochondrial oxidative stress mediates hypoxia

31.10. the imbalance in the circuitries of ros production and removal leads to impairment of cell signalling, oxidative damage of cell nents, .

mild oxidative stress induced by a low dose of cisplatin contributes

mitochondrial events involving hypoxiainduced drug resistance were assessed. hypoxia significantly reduced the cisplatininduced apoptosis of tcc cells.

[pdf] cisplatin resistance and redox

23.03. therefore, we hypothesized that the cisplatin resistance of ovarian cancer is linked to the ability to escape from traılmediated apoptosis. we .

failure of elevating calcium ınduces oxidative stress tolerance

09.07.2021 cisplatininduced toxicity involves various mechanisms, such as dna adduct formation, mitochondrial dysfunction, and oxidative stress [4] the .

oxidative stress after cisplatin treatment. the effect of dosage after.

01.10. objective: to investigate the role of oxidative stress in the development of cisplatin resistance in epithelial ovarian cancer eoc. methods: .

a reactive oxygen species scoring system predicts cisplatin

05.08.2021 pdf cisplatin is a commonly used chemotherapeutic drug, used for the treatment of malignant ovarian cancer, but acquired resistance limits .

[pdf] ginseng extract and its constituents alleviate cisplatin toxicity and

download scientific diagram oxidative stress after cisplatin treatment. cell lines could partially elucidate the development of cisplatin resistance.

wayne state university dissertations

08.11. ros status is associated with chemotherapy resistance. elevated oxidative stress and delicate redox balance were detected in cancer .

can pycnogenol prevent cisplatin

16.12. toxicity and reverse cisplatin resistance drug resistance of cisplatin oxidative stress is also implicated for cisplatininduced.

the role of oxidative stress ın the establishment of resistance to

belotte, jimmy, "the role of oxidative stress ın the establishment of resistance to cisplatin ın epithelial ovarian cancer cells" . wayne state university .

[pdf] lncrna mır4435

pycnogenol is a potent antioxidant that is used in medical conditions caused by oxidative stress. the aim of our study is to demonstrate the effects of .

loss of scribble confers cisplatin resistance during nsclc

known specific snps in oxidant and antioxidant genesalter their the role of oxidative stress in the development of cisplatin resistance in .

glaucocalyxin b attenuates ovarian cancer cell growth

24.11.2020 furthermore, nrf2 and ho1 mrna levels, as critical mole cules in the oxidative stress pathway, were inhibited by sirnas targeting mır44352hg,

knock

05.09. chemotherapeutic agents generate reactive oxygen species ros in patients undergoing cancer therapy, in whom toxically oxidative stress is .

melatonin ameliorates cisplatin

25.02.2022 cisplatin is the firstline chemotherapy drug for the clinical growth and cisplatin resistance ın vitro via activating oxidative stress

sodium arsenite ± hyperthermia sensitizes p53

involved in causing cisplatin resistance in ovarian cancer. cause oxidative stress such as dna damage, leading to cell death 12, 13.

suppression of sesn1 reduces cisplatin and hyperthermia

melatonin, which has the ability to provide resistance against oxidation, ameliorates cisplatininduced asthenozoospermia via reducing oxidative stress.

[pdf] oxidative metabolism drives inflammation

resistance decreases the effectiveness of cisplatin. like hyperthermia, arsenic causes oxidative stress and mitotic catastrophe taylor et al., .

cisplatin

09.10. however, some head and neck cancers have cisplatin resistance, for ros suppression and which protect cells from oxidative stress, .

ınositol

20.05. silencing induced resistance to cisplatin, resistant cells showed increased oxidative metabolism compared with their sensitive.

exploiting ros and metabolic differences to kill cisplatin resistant

results: hypoxia exacerbated the cisplatin resistance in lung cancer cells due to the increased expression of pkm2 that was oxidative stress detection.